A Novel and Powerful Way to Support Bone Health—Myostatin Dampening

A Novel and Powerful Way to Support Bone Health—Myostatin Dampening

Myostatin Dampening

Myostatin inhibition and increased protein synthesis play significant roles in bone health, particularly in the context of osteopenia and osteoporosis, with growing scientific literature supporting their therapeutic potential in musculoskeletal disorders.1 2

Clinical use of Fortetropin, derived from raw, fertile egg yolk powder, is a bioactive complex that helps reduce myostatin levels. Fortetropin increases lean body mass, reduces markers of protein breakdown, consistent with myostatin inhibition, and increases mTOR signaling, which is associated with muscle growth.3 4 5 Aging comes with many challenges, including a trend toward deleterious increases in myostatin, which contribute to challenges in maintaining a biological younger, more resilient body.

Myostatin and Bone Metabolism

Myostatin, encoded by the MSTN gene, is a myokine that primarily inhibits muscle growth but also affects bone metabolism. Excessive myostatin signaling contributes to muscle wasting and reduced bone mass through two key mechanisms: suppression of protein synthesis and activation of bone-resorbing osteoclasts. In skeletal diseases like osteopenia and osteoporosis, elevated myostatin has been linked to increased osteoclast differentiation and RANKL activation, leading to bone resorption exceeding bone formation. 6 7 In vitro, myostatin inhibits osteoblast differentiation and stimulates osteoclast activity in a dose-dependent manner. Mice deficient in myostatin also have decreased osteoclast numbers, increased cortical thickness, cortical tissue mineral density in the tibia, and increased vertebral bone mineral density. 8

Myostatin Inhibition in Osteopenia and Osteoporosis

Targeting myostatin has emerged as a promising strategy to counteract muscle and bone loss. Both animal and human studies demonstrate that myostatin inhibition benefits bone turnover and regeneration. Pharmacologic agents such as follistatin and myostatin-neutralizing antibodies have shown marked increases in osteogenic differentiation, bone calcification, and bone quality, particularly in models of osteoporosis and impaired bone healing. These effects are mediated via enhanced osteoblast activity, reduced osteoclastogenesis, and downregulation of pro-resorptive signals such as SMAD2/3.9

Transgenic and knockout mouse models deficient in myostatin exhibit increased muscle mass, higher bone mineral density, and enhanced stem cell-driven bone formation. Clinical interest in myostatin inhibitors is growing among elderly patients to address concurrent muscle and bone loss, offering dual protection against sarcopenia and osteoporosis.10

Protein Synthesis and Bone Health

Protein synthesis is equally vital for maintaining bone integrity and promoting bone formation. Adequate dietary protein supports optimal bone mass, particularly in environments of sufficient calcium and vitamin D intake. Increased protein intake elevates anabolic markers, such as IGF-1, which in turn boosts osteoblast activity and new bone formation.11

Synergistic Benefits

Inhibiting myostatin and promoting protein synthesis offer synergistic benefits for bone and muscle health, especially in aging populations at risk for osteopenia and osteoporosis. Myostatin inhibition not only curbs bone loss and muscle wasting but also supports anabolic processes essential for bone regeneration and strength, while increased dietary and endogenous protein synthesis fosters bone matrix development and maintenance.10

Clinical Take Home and Considerations:

A dynamic and powerful 2-in-1 approach to support muscle and bone health. Maintaining muscle mass and promoting protein synthesis are processes linked to bone health, as bones and muscles work together. Inhibiting myostatin promotes protein synthesis, reduces muscle breakdown, and supports healthy bones. An effective way to reduce excess myostatin is to use Fortetropin, an ingredient derived from raw, fertile egg yolk powder. Incorporating Fortetropin can further enhance the effectiveness of your existing muscle and bone protocol, when combined with targeted exercise and quality sleep, supporting the pursuit of strong muscles and bones well into the golden years.

 

References

1 Myostatin--the holy grail for muscle, bone, and fat? Curr Osteoporos Rep. 2013 Dec;11(4):407-14. doi: 10.1007/s11914-013-0160-5.
2 Myostatin Inhibitors: Panacea or Predicament for Musculoskeletal Disorders? J Bone Metab. 2020 Aug 31;27(3):151–165. doi: 10.11005/jbm.2020.27.3.151 3 Sharp MH, Lowery RP, Mobley CB, et al. The Effects of Fortetropin Supplementation on Body Composition, Strength, and Power in Humans and Mechanism of Action in a Rodent Model. J Am Coll Nutr. 2016;35(8):679-691. 4 Evans W, Shankaran M, Nyangau E, et al. Effects of Fortetropin on the Rate of Muscle Protein Synthesis in Older Men and Women: A Randomized, Double-Blinded, Placebo-Controlled Study. J Gerontol A Biol Sci Med Sci. 2021;76(1):108-114. 5 White DA, Harkin KR, Roush JK, Renberg WC, Biller D. Fortetropin inhibits disuse muscle atrophy in dogs after tibial plateau leveling osteotomy. PLoS One. 2020;15(4):e0231306.
6 Zhi X, Chen Q, Song S, Gu Z, Wei W, Chen H, Chen X, Weng W, Zhou Q, Cui J, Cao L., Myostatin Promotes Osteoclastogenesis by Regulating Ccdc50 Gene Expression and RANKL-Induced NF-κB and MAPK Pathways. Front Pharmacol. 2020 Nov 26;11:565163. doi: 10.3389/fphar.2020.565163
7 Baig MH, Ahmad K, Moon JS, Park SY, Lim JH, Chun HJ, Qadri AF, Hwang YC, Jan AT, Ahmad SS, Ali S, Shaikh S, Lee EJ, Choi I., Myostatin and its Regulation: A Comprehensive Review of Myostatin Inhibiting Strategies. Front Physiol. 2022 Jun 23;13:876078. doi: 10.3389/fphys.2022.876078 8 Deciphering Myostatin’s Regulatory, Metabolic, and Developmental Influence in Skeletal Diseases. Front. Genet. 12:662908. https://doi.org/10.3389/fgene.2021.662908 9 The elusive role of myostatin signaling for muscle regeneration and maintenance of muscle and bone homeostasis. Osteoporos Sarcopenia. 2023 Mar 27;9(1):1–7. doi: 10.1016/j.afos.2023.03.008 10 Omosule CL, Phillips CL Deciphering Myostatin’s Regulatory, Metabolic, and Developmental Influence in Skeletal Diseases. Front Genet. 2021;12. https://doi.org/10.3389/fgene.2021.662908 11 Li GB, Zhang L, Wang DE, AlQudsy L, Jiang JX, Xu HY, Shang P., Muscle-bone crosstalk and potential therapies for sarco-osteoporosis. Muscle-bone crosstalk and potential therapies for sarco-osteoporosis. J Cell Biochem. 2019; 120(9):14262-14273. https://doi.org/10.1002/jcb.28946
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